Supplementary Powders – BEHEMOTH LABZ

Synephrine HCl Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:40 +0000

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Synephrine HCl is the hydrochloride salt form of synephrine, a sympathomimetic compound associated primarily with bitter orange (Citrus aurantium). It has been studied for metabolic activity, energy expenditure, exercise-related applications, and weight-management research.

Product Information

Most human research involving synephrine concerns p-synephrine, the principal protoalkaloid associated with bitter orange. Research has examined its effects on resting metabolic rate, energy expenditure, cardiovascular parameters, and body composition.

How It Works

p-Synephrine interacts with adrenergic pathways involved in metabolic signaling and energy expenditure. Although structurally related to ephedrine, its pharmacological profile is different and should not be treated as interchangeable with ephedrine-type stimulants.

POTENTIAL BENEFITS

1. Supports Metabolic Activity

Human studies have found that p-synephrine can increase resting metabolic rate and energy expenditure. This makes it particularly relevant to research into metabolism and weight management.

2. Supports Energy Expenditure

Synephrine has demonstrated measurable effects on energy expenditure in human research. The magnitude of the effect varies according to dose, formulation, and whether it is combined with other stimulants.

3. Supports Weight-Management Research

Synephrine-containing products have been investigated extensively for weight management. Evidence is mixed, however, and systematic review data have not established a significant long-term weight-loss effect.

4. Exercise-Related Research Interest

Synephrine has also been studied in sports and exercise settings because of its effects on metabolic activity and adrenergic signaling.

Dosage

Human studies have commonly investigated p-synephrine in the approximate range of 10-53 mg/day, including studies lasting several weeks. These research amounts should not be interpreted as a universal recommended dose for Synephrine HCl products.

Side Effects

Potential concerns include increased blood pressure, increased heart rate, headache, nervousness, and cardiovascular stimulation. Combining synephrine with caffeine or other stimulants may increase cardiovascular stress.

References

Stohs SJ, Preuss HG, Shara M. A review of the human clinical studies involving Citrus aurantium (bitter orange) extract and its primary protoalkaloid p-synephrine. Int J Med Sci. 2012;9(7):527-538. PubMed
Stohs SJ, et al. The Safety and Efficacy of Citrus aurantium (Bitter Orange) Extracts and p-Synephrine: A Systematic Review and Meta-Analysis. PubMed

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Bromantane Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:39 +0000

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Bromantane is an adamantane-derived actoprotector developed in Russia and investigated for its ability to support physical and mental performance under demanding conditions. It has also been studied for psychophysiological effects and fatigue resistance.

Product Information

Bromantane belongs to the actoprotector class, compounds designed to improve resistance to physical stress without simply producing conventional stimulant effects. Human and animal research has investigated its effects on functional performance, fatigue, and adaptation.

How It Works

Bromantane appears to influence several central and metabolic pathways involved in adaptation and performance. Its pharmacology is distinct from classic stimulants, with research suggesting effects on dopaminergic systems and adaptive responses to stress.

POTENTIAL BENEFITS

1. Supports Physical Performance

Bromantane was developed as an actoprotector intended to improve physical work capacity and resistance to demanding conditions. Research has investigated its effects during fatigue, heat, hypoxia, and other physical stressors.

2. Supports Fatigue Resistance

One of Bromantane's defining research characteristics is its ability to improve performance under conditions associated with fatigue. Animal studies have demonstrated increased working capacity and improved recovery from experimentally induced fatigue.

3. Mental Performance Research

Human research has examined Bromantane's influence on EEG and psychophysiological performance, including studies in healthy volunteers showing favorable effects on functional state.

4. Adaptation to Environmental Stress

Bromantane has been studied under challenging environmental conditions such as heat and hypoxia. Historical human research reported improved resistance to combined environmental stressors following oral administration.

Dosage

Historical human research has included 250 mg orally in healthy volunteers and in environmental-stress experiments. These are study-specific historical doses rather than a modern standardized therapeutic regimen.

Side Effects

Potential effects may include stimulation, insomnia, headache, changes in mood, gastrointestinal symptoms, or alterations in blood pressure and heart rate. Long-term safety information outside historical clinical use is limited.

References

Kim H, et al. The pharmacology of actoprotectors: practical application for improvement of mental and physical performance. Pharmacol Ther. 2013. PMID: 24009833. PubMed
Viatleva OA, et al. The neuro- and psychophysiological effects of bromantane. Voen Med Zh. 2000. PMID: 10998997. PubMed
Sedov AV, et al. Experimental rationale for the use of drugs to increase the resistance of the human body to the combined action of carbon monoxide and hyperthermia. Med Tr Prom Ekol. 1993. PMID: 8087458. PubMed

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NAD+ Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:39 +0000

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NAD+ (nicotinamide adenine dinucleotide) is a naturally occurring coenzyme involved in cellular energy production, redox reactions, DNA repair, and metabolic signaling. NAD+ has become an important area of research surrounding cellular health, aging, mitochondrial function, and metabolic resilience.

Product Information

NAD+ is the oxidized form of nicotinamide adenine dinucleotide and is present throughout the body's cells. It participates in mitochondrial energy production and serves as a substrate for enzymes including sirtuins and PARPs. Direct NAD+ administration has been studied in humans, although the clinical evidence remains considerably smaller than the broader literature on NAD+ precursors such as nicotinamide riboside and NMN.

How It Works

NAD+ helps cells transfer energy during metabolic reactions and supports enzymes involved in cellular maintenance and stress responses. Its availability can influence mitochondrial metabolism, DNA-repair pathways, and cellular signaling.

POTENTIAL BENEFITS

1. Supports Cellular Energy Production

NAD+ is central to mitochondrial energy metabolism and helps cells convert nutrients into usable energy. Maintaining NAD+ availability is therefore closely tied to normal cellular energy production.

2. Supports Healthy Aging Research

NAD+ participates in DNA repair, cellular stress responses, and sirtuin signaling. These roles have made NAD+ one of the most closely studied molecules in modern longevity and healthy-aging research.

3. Supports Metabolic Function

NAD+-dependent enzymes contribute to glucose, fatty-acid, and mitochondrial metabolism. Researchers continue to investigate whether maintaining NAD+ availability can support metabolic resilience as people age.

4. Supports Cellular Maintenance

NAD+ serves as a substrate for enzymes involved in DNA-damage responses and other cellular maintenance processes. This has made NAD+ particularly interesting in research on age-related cellular decline.

Dosage

There is no universally established clinical dosage for direct NAD+ supplementation for general wellness or longevity. A human pilot study examined a continuous IV infusion at 3 umol/min for six hours, demonstrating rapid metabolism of infused NAD+. This was a pharmacokinetic research study rather than an established general supplementation regimen.

Side Effects

Human safety data for direct NAD+ administration remain limited. Infusion-related symptoms have been reported in clinical practice, and direct IV administration should be performed only within an appropriate medical setting.

References

Grant R, et al. A Pilot Study Investigating Changes in the Human Plasma and Urine NAD+ Metabolome During a 6 Hour Intravenous Infusion of NAD. Front Aging Neurosci. 2019;11:257. PubMed

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Noopept (Omberacetam) Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:38 +0000

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Noopept, also known as Omberacetam or GVS-111, is a synthetic proline-containing dipeptide ester developed as a potent nootropic compound. It has been investigated for memory, cognitive impairment, neuroprotection, and adaptation to physical stress.

Product Information

Noopept has been studied in Russian clinical literature as a cognitive and neuroprotective agent. Human studies have used 10-20 mg/day, while preclinical research has explored mechanisms involving neurotrophic signaling, oxidative stress, inflammation, and glutamate-related neurotoxicity.

How It Works

Noopept appears to influence multiple neuronal pathways rather than acting through a single receptor. Research has associated it with neurotrophic signaling, antioxidant activity, modulation of glutamate-related toxicity, and effects on memory consolidation and retrieval.

POTENTIAL BENEFITS

1. Supports Memory Research

Noopept has been investigated for memory acquisition, consolidation, and retrieval. Human clinical research has reported improvements in selected cognitive measures in people with cognitive impairment.

2. Neuroprotective Research

Preclinical research has reported protection against ischemic and excitotoxic neuronal damage. These findings have contributed to Noopept's reputation as a neuroprotective as well as nootropic compound.

3. Cognitive-Function Support

Clinical studies have examined Noopept in patients with cognitive impairment associated with stroke, vascular disease, and trauma. A published study used 20 mg/day for two months and reported improved cognitive measures.

4. Anxiolytic and Adaptogenic Research

Noopept has also been investigated for anxiety-related effects and adaptation to environmental stress. Human volunteer research reported effects on psychological and physical performance under challenging environmental conditions.

Dosage

Published human studies have used 10 mg twice daily (20 mg/day), including a clinical study involving patients with stroke-related cognitive impairment. Earlier clinical-development literature also describes 5 mg and 10 mg tablet strengths. These doses reflect historical clinical research rather than a recommendation for unsupervised use.

Side Effects

Reported clinical studies suggest relatively good short-term tolerability, but possible headache, irritability, sleep changes, gastrointestinal effects, or other CNS symptoms may occur. Long-term safety data remain limited, especially outside the populations studied clinically.

References

Gudasheva TA, et al. The original novel nootropic and neuroprotective agent noopept. Eksperimental'naia i klinicheskaia farmakologiia. 2002. PMID: 12596521. PubMed
Amelin AV, Iliukhina AIu, Shmonin AA. Noopept in the treatment of mild cognitive impairment in patients with stroke. Zh Nevrol Psikhiatr Im S S Korsakova. 2011;111(10 Pt 1):44-46. PMID: 22500312. PubMed
Shabanov PD, Ganapol'skii VP, Aleksandrov PV. Meteoadaptogenic properties of peptide drugs in healthy volunteers. Eksperimental'naia i klinicheskaia farmakologiia. 2007;70(6):41-47. PMID: 18318195. PubMed
Szeleszczuk L, et al. Physicochemical and structural analysis of N-phenylacetyl-L-prolylglycine ethyl ester (Noopept). 2024. PMID: 39298839. PubMed

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NSI-189 Free Base Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:37 +0000

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NSI-189 Free Base is the non-phosphate form of NSI-189, a small-molecule compound developed to investigate neurogenesis and hippocampal function. The active NSI-189 molecule is the basis of the clinical research conducted with its phosphate salt.

Product Information

The clinical development of NSI-189 primarily used NSI-189 phosphate, including Phase 1B and Phase 2 randomized trials. The free-base form represents the same active molecular entity without the phosphate salt, but its pharmacokinetic and formulation characteristics should not automatically be assumed identical.

How It Works

NSI-189 was developed as a neurogenic compound intended to influence neuronal growth and hippocampal function. Unlike traditional antidepressants, its proposed activity does not center on direct monoamine reuptake inhibition.

POTENTIAL BENEFITS

1. Neurogenesis Research

NSI-189 is particularly notable for its focus on neurogenesis and hippocampal biology. This mechanism has made it an important research compound for investigating how neuronal growth may influence cognition and mood.

2. Cognitive Research

Clinical trials included cognitive and physical-functioning assessments, with some exploratory improvements observed during early development.

3. Mood-Related Research

NSI-189 was investigated in patients with major depressive disorder. Although Phase 2 results did not establish superiority on the primary depression endpoint, some secondary outcomes showed potential activity.

4. Hippocampal Function

The compound's development was based in part on the relationship between hippocampal neurogenesis and cognitive and emotional function, giving NSI-189 a distinctive neuroscience research profile.

Dosage

Published human trials used NSI-189 phosphate, not a separately validated free-base dosing protocol. Phase 2 studies tested 40 and 80 mg/day of the phosphate form. These doses should not automatically be treated as equivalent dosing for a free-base formulation.

Side Effects

The human safety profile is limited to clinical development of NSI-189 phosphate. Potential CNS and systemic adverse effects remain incompletely characterized for separately formulated free-base material.

References

Fava M, et al. A Phase 1B, randomized, double blind, placebo-controlled, multiple-dose escalation study of NSI-189 phosphate, a neurogenic compound, in depressed patients. Mol Psychiatry. 2016;21(10):1372-1380. PMID: 26643541. PubMed
Papakostas GI, et al. A phase 2, double-blind, placebo-controlled study of NSI-189 phosphate, a neurogenic compound, among outpatients with major depressive disorder. Mol Psychiatry. 2020;25(7):1569-1579. PMID: 30626911. PubMed

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NSI-189 Phosphate Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:36 +0000

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NSI-189 Phosphate is a small-molecule neurogenic compound developed to investigate hippocampal neurogenesis and cognitive and mood-related disorders. It progressed through human clinical trials in major depressive disorder.

Product Information

NSI-189 was evaluated in randomized controlled clinical studies using oral doses of 40 and 80 mg/day. Phase 1B research suggested potential effects on depressive and cognitive measures, while the subsequent Phase 2 trial did not significantly improve its primary depression endpoint.

How It Works

NSI-189 was designed to influence neurogenesis, particularly within the hippocampus. Its mechanism differs from traditional antidepressants because it does not primarily depend on monoamine reuptake inhibition.

POTENTIAL BENEFITS

1. Neurogenesis Research

NSI-189 was developed specifically around the concept of stimulating neuronal growth and hippocampal neurogenesis, making it a distinctive compound in neuroscience research.

2. Cognitive Function Research

Human studies included cognitive and physical-functioning assessments, with Phase 1B research reporting exploratory improvements in selected cognitive measures.

3. Mood Research

NSI-189 progressed into clinical trials for major depressive disorder. Although the primary Phase 2 depression endpoint was not statistically significant, several secondary measures showed signals of potential activity.

4. Non-Monoaminergic Approach

NSI-189's proposed neurogenic mechanism differentiates it from conventional antidepressants that primarily target serotonin, norepinephrine, or dopamine reuptake.

Dosage

Human Phase 1B research evaluated 40 mg once daily, 40 mg twice daily, and 40 mg three times daily. A later Phase 2 trial evaluated 40 mg/day and 80 mg/day for 12 weeks. These are clinical-trial doses rather than an established current treatment regimen.

Side Effects

Clinical trials reported generally acceptable short-term tolerability, although adverse events occurred. Because NSI-189 has not become an established mainstream therapy, long-term safety and broader drug-interaction data remain limited.

References

Fava M, et al. A Phase 1B, randomized, double blind, placebo-controlled, multiple-dose escalation study of NSI-189 phosphate, a neurogenic compound, in depressed patients. Mol Psychiatry. 2016;21(10):1372-1380. PMID: 26643541. PubMed
Papakostas GI, et al. A phase 2, double-blind, placebo-controlled study of NSI-189 phosphate, a neurogenic compound, among outpatients with major depressive disorder. Mol Psychiatry. 2020;25(7):1569-1579. PMID: 30626911. PubMed

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RU58841 Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:35 +0000

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RU58841 is a nonsteroidal antiandrogen developed for topical inhibition of androgen-receptor activity. It has attracted considerable research interest in androgen-dependent skin and hair conditions, particularly androgenetic alopecia.

Product Information

RU58841 competes with androgens at the androgen receptor. Experimental research has demonstrated suppression of androgen-dependent signaling in human prostate cells and activity against hair-growth effects in animal models.

How It Works

RU58841 acts as an androgen-receptor antagonist, reducing the ability of testosterone-derived androgens such as DHT to activate the receptor. Its intended research application has primarily involved local rather than systemic androgen blockade.

POTENTIAL BENEFITS

1. Androgen-Receptor Research

RU58841 directly targets androgen-receptor signaling, making it a useful research compound for understanding how androgen activity affects skin, hair follicles, and other androgen-responsive tissues.

2. Hair-Loss Research

Animal studies have investigated RU58841 as a topical antiandrogen for androgen-dependent hair loss. Its local receptor activity is the principal reason it has become associated with hair-regrowth research.

3. DHT-Signaling Modulation

Rather than reducing testosterone production directly, RU58841 works downstream by interfering with androgen-receptor activation. This provides a mechanistically distinct approach to androgen signaling.

4. Nonsteroidal Antiandrogen

RU58841 is structurally distinct from steroidal antiandrogens. Laboratory studies demonstrated competitive suppression of DHT-driven androgen-receptor activation.

Dosage

There is no established human therapeutic dosage for RU58841. Historical research has focused primarily on topical experimental administration, including animal models. Online dosing practices should not be presented as clinically validated human dosing.

Side Effects

Human safety data are inadequate. Potential concerns include local irritation and unpredictable systemic antiandrogenic effects if absorption occurs. The long-term safety of unsupervised topical use has not been established.

References

Sintov A, et al. Evaluation of RU58841 as an anti-androgen in prostate PC3 cells and a topical anti-alopecia agent in the bald scalp of stump-tailed macaques. Endocrine. 1998;9(1):39-43. PMID: 9798729. PubMed
Miyamoto H, et al. Promotion of agonist activity of antiandrogens by the androgen receptor coactivator, ARA70, in human prostate cancer DU145 cells. Proc Natl Acad Sci USA. 1998;95(13):7379-7384. PMID: 9636157. PubMed

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Dihexa Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:35 +0000

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Dihexa is a small-molecule analog derived from angiotensin IV research and developed as a blood-brain-barrier-penetrant HGF/c-Met pathway modulator. It has attracted substantial interest because of its reported effects on synaptic formation and cognitive performance in animal models.

Product Information

Research has shown that Dihexa binds hepatocyte growth factor (HGF), enhances HGF-dependent c-Met signaling, and promotes hippocampal spinogenesis and synaptogenesis. Oral Dihexa has also demonstrated procognitive effects in animal models.

How It Works

Dihexa interacts with HGF and enhances signaling through the c-Met receptor. This pathway is involved in neuronal growth, synaptic development, and cellular repair, providing the mechanistic basis for Dihexa's cognitive research profile.

POTENTIAL BENEFITS

1. Synaptogenesis Research

Dihexa has demonstrated the ability to promote hippocampal spinogenesis and synaptogenesis in experimental systems. These effects make it particularly interesting in research focused on forming and strengthening neuronal connections.

2. Cognitive Performance Research

Orally administered Dihexa has produced procognitive effects in animal models, including improvements in spatial-learning tasks. This is one of the compound's most notable research characteristics.

3. Neuroplasticity Research

By influencing HGF/c-Met signaling, Dihexa may affect pathways involved in neuronal growth and structural plasticity. This gives it a broader research profile than compounds acting solely as acute stimulants.

4. Blood-Brain-Barrier Penetration

Dihexa was specifically developed to overcome the blood-brain-barrier limitations associated with earlier angiotensin-derived molecules, allowing it to reach central nervous system targets in experimental models.

Dosage

There is no established human dosage for Dihexa. Research doses in animal and cellular models should not be interpreted as human therapeutic dosing.

Side Effects

Human safety data are very limited. Because HGF/c-Met signaling regulates growth, differentiation, and tissue remodeling, unintended effects from sustained pathway activation remain an important research concern.

References

McCoy AT, et al. The procognitive and synaptogenic effects of angiotensin IV-derived peptides are dependent on activation of the hepatocyte growth factor/c-met system. J Pharmacol Exp Ther. 2014;349(2):286-293. PMID: 25187433. PubMed
Mizuno S, Nakamura T. The Brain Hepatocyte Growth Factor/c-Met Receptor System: A New Target for the Treatment of Alzheimer's Disease. Front Pharmacol. 2015. PMID: 25649658. PubMed

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7,8-Dihydroxyflavone Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:34 +0000

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7,8-Dihydroxyflavone (7,8-DHF) is a flavone derivative best known for its activity at the TrkB receptor, the principal receptor for brain-derived neurotrophic factor (BDNF). It has become a prominent research compound in neuroscience and neuroprotection.

Product Information

Preclinical research has shown that 7,8-DHF can activate TrkB and downstream signaling pathways associated with neuronal survival, differentiation, and synaptic plasticity. Studies have examined its effects in models of neurodegeneration, stroke, Parkinsonism, and cognitive dysfunction.

How It Works

7,8-DHF binds to and activates TrkB, mimicking certain signaling effects normally produced by BDNF. TrkB activation can influence neuronal survival, synaptic plasticity, and neurotrophic signaling.

POTENTIAL BENEFITS

1. Supports Neurotrophic Signaling

7,8-DHF directly activates TrkB-related signaling, making it particularly interesting for research into BDNF-associated neuronal function.

2. Memory and Cognitive Research

Animal studies have reported improvements in memory-related behavior following 7,8-DHF administration, supporting its investigation as a potential cognitive research tool.

3. Neuroprotective Potential

7,8-DHF has protected neurons in several experimental models, including models involving excitotoxicity, ischemic injury, and dopaminergic degeneration.

4. Synaptic Plasticity Research

TrkB activation is closely associated with synaptic remodeling and plasticity. Research has demonstrated that 7,8-DHF can promote signaling linked to synaptogenesis and neuronal maturation.

Dosage

There is no established human therapeutic dosage for 7,8-DHF. Published doses are primarily from cellular and animal experiments and should not be treated as validated human dosing.

Side Effects

Human safety data are limited. Because TrkB signaling influences numerous neuronal and cellular processes, the consequences of sustained pharmacological activation remain incompletely characterized.

References

Liu X, et al. Biochemical and biophysical investigation of the brain-derived neurotrophic factor mimetic 7,8-dihydroxyflavone in the binding and activation of the TrkB receptor. J Biol Chem. 2014;289(40):27571-27584. PMID: 25143381. PubMed
Jang SW, et al. A selective TrkB agonist with potent neurotrophic activities by 7,8-dihydroxyflavone. Proc Natl Acad Sci USA. 2010;107(6):2687-2692. PMID: 20133810. PubMed
Liu C et al. 7,8-dihydroxyflavone protects 6-OHDA and MPTP induced dopaminergic neuron degeneration through activation of TrkB in rodents. Neurosci Lett. 2016. PMID: 27019033. PubMed

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Amentoflavone (20% Extract) Powder

Zeeshan Tariq — Fri, 11 Sep 2026 16:12:33 +0000

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Amentoflavone is a naturally occurring biflavonoid found in several medicinal plants, including Selaginella species. A standardized 20% extract provides a concentrated source of Amentoflavone for nutritional and research applications.

Product Information

Amentoflavone has been extensively investigated for antioxidant, anti-inflammatory, metabolic, neurological, and cellular effects. Despite its broad preclinical profile, poor water solubility and limited bioavailability have restricted its development as a conventional therapeutic drug.

How It Works

Amentoflavone influences multiple molecular pathways, including inflammatory signaling, oxidative-stress pathways, and cellular kinase systems. It is therefore considered a multi-target flavonoid rather than a compound acting through a single receptor.

POTENTIAL BENEFITS

1. Antioxidant Activity

Amentoflavone has demonstrated significant antioxidant activity in laboratory research, making it relevant to studies involving oxidative stress and cellular protection.

2. Anti-Inflammatory Research

Research has identified effects on inflammatory pathways, including NF-kB and related signaling networks. This broad activity has contributed to substantial interest in Amentoflavone research.

3. Neuroprotective Research

Amentoflavone has been investigated in neurological models involving oxidative stress, inflammation, neuronal signaling, and cellular survival, creating a broad neuroprotective research profile.

4. Metabolic Research

Experimental research has examined Amentoflavone in relation to glucose and lipid metabolism, including pathways relevant to metabolic dysfunction and healthy aging.

Dosage

There is no established human therapeutic dosage for Amentoflavone. A 20% extract is formulation-specific, so the actual Amentoflavone content depends on the total extract quantity. Human dosing should not be inferred from animal studies.

Side Effects

Human safety data are limited. Amentoflavone also has demonstrated the ability to inhibit several drug-metabolizing enzymes in laboratory studies, creating potential drug-interaction concerns.

References

Yu S, Yan H, Zhang L, et al. A Review on the Phytochemistry, Pharmacology, and Pharmacokinetics of Amentoflavone, a Naturally-Occurring Biflavonoid. Molecules. 2017;22(2):299. PMID: 28212342. PubMed
Zhang X, et al. Insights Into Amentoflavone: A Natural Multifunctional Biflavonoid. Front Pharmacol. 2022. PMID: 35002708. PubMed
Wang Y, et al. Amentoflavone for treating cardiocerebrovascular diseases and neurological disorders. Front Pharmacol. 2024. PMID: 39624841. PubMed

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